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AR cooperates with SMAD4 to maintain skeletal muscle homeostasis.

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Forouhan M. et al, (2022), Acta Neuropathol

Proof of concept of peptide-linked blockmiR-induced MBNL functional rescue in myotonic dystrophy type 1 mouse model

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Overby SJ. et al, (2022), Molecular Therapy - Nucleic Acids, 27, 1146 - 1155

Control of backbone chemistry and chirality boost oligonucleotide splice switching activity.

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Kandasamy P. et al, (2022), Nucleic Acids Res

Enhancing the Therapeutic Potential of Extracellular Vesicles Using Peptide Technology


Martin Perez C. et al, (2022), Methods in Molecular Biology, 2383, 119 - 141

Preclinical characterization of antagomiR-218 as a potential treatment for myotonic dystrophy.

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Cerro-Herreros E. et al, (2021), Mol Ther Nucleic Acids, 26, 174 - 191

Dystrophin involvement in peripheral circadian SRF signalling

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Betts CA. et al, (2021), Life Science Alliance, 4, e202101014 - e202101014

Extracellular vesicles as a next-generation drug delivery platform

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WOOD M. et al, (2021), Nature Nanotechnology

Emerging Oligonucleotide Therapeutics for Rare Neuromuscular Diseases.

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Aoki Y. and Wood MJA., (2021), J Neuromuscul Dis

Improving assessments of data-limited populations using life-history theory

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Horswill C. et al, (2021), Journal of Applied Ecology, 58, 1225 - 1236

Applications of digital imaging and analysis in seabird monitoring and research

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Edney AJ. and Wood MJ., (2021), Ibis, 163, 317 - 337

Targeting the 5' untranslated region of SMN2 as a therapeutic strategy for spinal muscular atrophy.

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Winkelsas AM. et al, (2021), Mol Ther Nucleic Acids, 23, 731 - 742

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