Evaluating inositol phospholipid interactions with inward rectifier potassium channels and characterising their role in disease
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New insights into KATP channel gene mutations and neonatal diabetes mellitus.
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Nucleotide inhibition of the pancreatic ATP-sensitive K+ channel explored with patch-clamp fluorometry.
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Erratum. Fetal Macrosomia and Neonatal Hyperinsulinemic Hypoglycemia Associated With Transplacental Transfer of Sulfonylurea in a Mother With KCNJ11-Related Neonatal Diabetes. Diabetes Care 2014;37:3333-3335
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Tamoxifen administration in pregnant mice can be deleterious to both mother and embryo.
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Diabetes causes marked inhibition of mitochondrial metabolism in pancreatic β-cells.
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Correction to: Q&A: insulin secretion and type 2 diabetes: why do β-cells fail?
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Hyperglycaemia adversely affects mitochondrial function in pancreatic islets
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Activation mechanism of ATP-sensitive K+ channels explored with real-time nucleotide binding.
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Insulin inhibits glucagon release by SGLT2-induced stimulation of somatostatin secretion.
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Low extracellular magnesium does not impair glucose-stimulated insulin secretion.
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Increased NEFA levels reduce blood Mg2+ in hypertriacylglycerolaemic states via direct binding of NEFA to Mg2.
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Dysregulation of Glucagon Secretion by Hyperglycemia-Induced Sodium-Dependent Reduction of ATP Production.
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Monitoring real-time hormone release kinetics via high-content 3-D imaging of compensatory endocytosis.
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Magnesium deficiency prevents high-fat-diet-induced obesity in mice.
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Permanent neonatal diabetes: combining sulfonylureas with insulin may be an effective treatment.
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Cardiac Dysfunction and Metabolic Inflexibility in a Mouse Model of Diabetes Without Dyslipidemia.
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FTO demethylase activity is essential for normal bone growth and bone mineralization in mice.
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