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Golgins have been implicated in the maintenance of Golgi architecture. Recent studies have shown, however, that mice lacking the golgin TRIP11/GMAP-210 have normal Golgi stacks, but show developmental problems related to defective cilium formation and function.

Original publication

DOI

10.1016/j.cub.2009.01.049

Type

Journal article

Journal

Curr Biol

Publication Date

24/03/2009

Volume

19

Pages

R253 - R255

Keywords

Animals, Carrier Proteins, Cilia, Flagella, Golgi Apparatus, Humans, Membrane Proteins, Mice, Nuclear Proteins, Protein Transport