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Persistence of latently infected CD4(+) T cell "reservoirs" presents a major obstacle for current HIV-1 antiretroviral therapies. In Cell Host & Microbe, two separate studies use model systems of latent infection to dissect the influence of host genes on HIV-1 transcription, yielding intriguing, though partially contradictory, conclusions.

Original publication

DOI

10.1016/j.chom.2008.07.010

Type

Journal article

Journal

Cell Host Microbe

Publication Date

14/08/2008

Volume

4

Pages

89 - 91

Keywords

CD4-Positive T-Lymphocytes, HIV Infections, HIV-1, Host-Pathogen Interactions, Humans, Transcription, Genetic, Virus Latency