Cookies on this website

We use cookies to ensure that we give you the best experience on our website. If you click 'Accept all cookies' we'll assume that you are happy to receive all cookies and you won't see this message again. If you click 'Reject all non-essential cookies' only necessary cookies providing core functionality such as security, network management, and accessibility will be enabled. Click 'Find out more' for information on how to change your cookie settings.

X chromosome inactivation triggered by Xist RNA can only occur in specific developmental contexts. In this issue of Developmental Cell, Agrelo et al. show that the nuclear matrix protein SATB1 is a critical determinant of Xist responsiveness.

Original publication

DOI

10.1016/j.devcel.2009.03.013

Type

Journal article

Journal

Dev Cell

Publication Date

04/2009

Volume

16

Pages

483 - 484

Keywords

Animals, Embryonic Stem Cells, Gene Silencing, Humans, Lymphoma, Matrix Attachment Region Binding Proteins, Mice, Models, Biological, RNA, Long Noncoding, RNA, Untranslated, X Chromosome Inactivation